Robbins Basic Pathology E-Book: Robbins Basic Pathology E-BookVinay Kumar, Abul K. Abbas, Jon C. Aster Elsevier Health Sciences, 8 mrt 2017 - 928 pagina's Part of the trusted Robbins and Cotran family, Robbins Basic Pathology provides a readable, well-illustrated and concise overview of the principles of human pathology that's ideal for today's busy students. This thoroughly revised edition continues with a strong emphasis on pathogenesis and the clinical features of disease, adding new artwork and more schematic diagrams to further aid in summarizing key pathologic processes and expand the already impressive illustration program.
|
Inhoudsopgave
| 1 | |
| 31 | |
3 Inflammation and Repair | 57 |
4 Hemodynamic Disorders Thromboembolism and Shock | 97 |
5 Diseases of the Immune System | 121 |
6 Neoplasia | 189 |
7 Genetic and Pediatric Diseases | 243 |
8 Environmental and Nutritional Diseases | 299 |
14 Kidney and Its Collecting System | 549 |
15 Oral Cavities and Gastrointestinal Tract | 583 |
16 Liver and Gallbladder | 637 |
17 Pancreas | 679 |
18 Male Genital System and Lower Urinary Tract | 691 |
19 Female Genital System and Breast | 713 |
20 Endocrine System | 749 |
21 Bones Joints and Soft Tissue Tumors | 797 |
Veelvoorkomende woorden en zinsdelen
abnormalities acid activation acute addition agents anemia antibodies antigens appear artery associated bacterial bind blood bone called cancer carcinoma cardiac cause cells cellular changes Chapter chromosome chronic clinical common complex contain cytokines cytoplasm damage death defects deficiency deposition discussed disease disorders drugs effects endothelial enzymes example expression extracellular factors failure fibrosis function genes genetic growth heart host human immune important increased individuals induce infarction infection inflammation inflammatory injury involved later lead lesions leukocytes levels liver loss lung lymphocytes lymphoma macrophages major malignant manifestations mechanisms mediators membrane microbes molecules muscle mutations myocardial infarction necrosis neoplasms normal occur organs pathways patients plasma platelet present primary produce progressive proliferation proteins pulmonary reactions receptors red cells release response result risk role seen severe signaling skin specific stem stimulate syndrome Table therapy tion tissue tumor typically usually vascular vessels viral wall